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Undeveloped embryonic and fetal defense systems are unable to counteract an overload of OS without support from exogenous antioxidants [282]

DON treatment activates AMPK in cancer cells by increasing the AMP/ATP ratio and decreasing the expression of c-Myc proteins, thereby inhibiting glycolytic metabolism and suppressing cancer cell growth in vitro 145

Adenosine and the concept of retaliatory metabolites

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A key consideration in the safety comparison is administration route

Physiological ROS levels are essential to neuronal functions, such as enhancing synaptic plasticity, long-term potentiation, and memory formation