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The kidney has been recognized as a main regulator of SUA and renal urate excretion is determined by the balance of the reabsortion and secretion of urates 2

M1, macrophage type 1

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Acetylcholine (ACh) released from neurons activates muscarinic ACh receptors (MRs) on ECs, increasing nitric oxide (NO) production, EC hyperpolarization, and consequently, elevated CBF [92]

(2012) Molecular characterization of the regenerative response induced by intrarenal transplantation of selected renal cells in a rodent model of chronic kidney disease Cells Tissues Organs, 196, 374384 25