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Excessive activation of MR leads to increased expression of pro-inflammatory and profibrotic genes, and generation of reactive oxygen species, contributory to tubular inflammation and fibrosis ( Overall, the activation of RAAS in DKD results in renal tubular injury through inflammation, hypoxia, fibrosis, and altered sodium and water handling

Acknowledgements We thank Prof Wangsen Cao for their critical review and thoughtful discussion of the manuscript

So basically what you're left with is very clean, very empty water

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