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The findings showed that using bispecific Ab (anti-EGFR and anti-c-Met) could suppress growth in anti-EGFR resistant cells more efficiently compared to anti-EGFR agents, such as gefitinib, erlotinib, and so on [149]

Together, these peptides are commonly discussed as complementary in research contextssupporting a broad, recovery-focused environment through multiple signalling pathways

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