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This outcome indicates that both molecules can interact cooperatively with either Keap1-NRF2 complex or Keap-1 monomer itself or HES-1 proteins, without exhibiting any antagonistic effects between the two compounds

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The lack of statistically significant reduction in infarct volume, despite a numerical decrease, suggests that DSIPs primary benefit in this model might be more related to functional neurorecovery and neural network plasticity rather than a direct, massive reduction of necrotic tissue [1]

No published study has reported Dihexa accelerating tumor growth, and no human cancer-incidence data exists in either direction