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Key Features Specifically formulated to target GLP-1, GIP, and Glucagon receptors for superior weight loss efficacy compared to single or dual agonists
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After RYGB, ghrelin (produced in the stomach) as well as duodenally produced gastric inhibitory peptide (GIP) and cholecystokinin are not consistently changed in the plasma in humans and rodent models
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Cardiac-related signals were also observed
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