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Erythropoietin inhibits SGK1-dependent TH17 induction and TH17-dependent kidney disease
Glucagon receptor co-agonism introduces the energy expenditure dimension, creating a two-front intervention
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References de Bie, P., Muller, P., Wijmenga, C
This effect is particularly important for: Wound healing Tendon and ligament repair (notoriously poor blood supply) Cardiac tissue recovery Gastrointestinal mucosal regeneration EGF (Epidermal Growth Factor) EGF promotes cell proliferation and differentiation, particularly in epithelial tissues