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An important mechanism in GIO pathogenesis is associated with increased osteclastogenesis resulting from activation of macrophage colony-stimulating factor (M-CSF) and receptor activator of nuclear factor-kappa B (NF-B) ligand (RANKL) [29]

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The EPR effect describes the phenomenon in which certain macromolecules of a specific sizesuch as liposomes, nanoparticles, and some macromolecular drugspermeate more readily into tumor tissues and are retained there longer compared to normal tissues

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In phase 2 and phase 3 trials, cagrilintide produced dosedependent weight loss with a predominantly gastrointestinal sideeffect profile [4] [5]