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Higher plasmatic SOD activity was also observed in MASLD subjects with high intrahepatic fat content (Monserrat-Mesquida et al

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A common homozygous deletion, known as the GSTT1 null genotype, removes the entire gene and leads to complete absence of GSTT1 protein and activity

In this review, we summarize the current existing literatures on NADPH homeostasis, including its biological functions, regulatory mechanisms and the corresponding therapeutic interventions in human cancers, providing insights into therapeutic implications of targeting NADPH metabolism and the associated mechanism for cancer therapy