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10.1002/dneu.22382 Dev

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In terms of intervention, aerobic exercise upregulates LONP1 and mitochondrial-encoded genes in aged mouse skeletal muscle, ameliorating mitochondrialnuclear signaling imbalance and activating the UPRmt, indicating that exercise may improve muscle dysfunction in aging or pathological states by enhancing LONP1 expression or activity [136]

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FMR1 CGG allele size and prevalence ascertained through newborn screening in the United States