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Primary, antagonistic, and integrative hallmarks link molecular damage to systemic aging, helping clarify where therapies like GLP-1s may have a positive influence and which physiological pathways they are most likely to influence [2,3]
doi: 10.1016/j.cej.2025.160596
Abstract Like other autoimmune diseases, rheumatoid arthritis (RA) develops in distinct stages, with each phase of disease linked to immune cell dysfunction
Rajiv SNewark Easy to use and helped us get some clear results
NO is an ephemeral but pleiotropic molecule
Additionally, small-molecule inhibitors, such as retinoic acid receptor-related orphan receptor gamma-t (RORt) inhibitors (e.g., VTP-43742) and tyrosine kinase 2 (TYK2) inhibitors (e.g., deucravacitinib), are currently in development
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