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Perturbation of glutathione homeostasis sensitized hepatocytes to TNF-induced cell death TNF plays an important role in the progression of NAFLD

High enough to be bacteriostatic (inhibiting bacterial growth) without being bactericidal (killing bacteria outright at first contact)

Compounds such as alpha-chloroacetamide-based inhibitors achieve this by covalently binding to a cysteine residue in the SAM-binding pocket of NNMT 202

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