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Glycyl-l-histidyl-l-lysine prevents copper- and zinc-induced protein aggregation and central nervous system cell death in vitro

Lobello et al., 2016), which might be due to irons induction of excess ROS (Zhang et al., 2020e)

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Furthermore, the value of such effects of BPC 157 reported [67] (which do not resemble the results obtained with any other serotonergic drug using this method [178,179,180,181]) is fully supported by the counteracting potential of BPC 157 therapy on serotonin syndrome [66]