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Importantly, Ca 2+ - and ROS-dependent LMP has been reported to contribute to necroptosis via release of cathepsins, cathepsins-mediated mitochondrial dysfunction, and lysosome-dependent regulation of RIPK1 and RIPK3 post-translational modifications [141]

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An important implication is that patients with ME/CFS and those with fatigue in the course of long COVID should not be studied as a single group, at least until the mechanisms are better understood

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