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The distinction separating theoretical risk from observed harm is significant: BPC-157 promotes physiological angiogenesis in the context of tissue injury repair rather than pathological angiogenesis associated with tumor microenvironments

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This is consistent with studies in experimental models of cancer, which correlate anti-angiogenic treatment with increased local tumor invasiveness and formation of distant metastasis [289,290,291,292]

Why PeptideClock uses first-order decay logic, what that means in practice, and where the model should be treated as an estimate rather than a measured assay