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Zaman GJ, Lankelma J, van Tellingen O, Beijnen J, Dekker H, Paulusma C et al

In addition, GSSG retained in mitochondria during oxidative stress can lead to the S-gluthionylation of target proteins with mitochondrial dysfunction (20, 21)

The Genetic Switch: Understanding Nrf2 Activation If glutathione is conceptualized as the primary fuel of the cellular antioxidant system, a crucial question concerns the regulatory engine that governs its synthesis and utilization

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Constant stress triggers the bodys fight or flight" response, which is controlled by the sympathetic nervous system and the hypothalamic-pituitary-adrenal axis

The observations from our animal studies prompted us to examine whether dysregulated cellular glutathione homeostasis could render hepatocytes susceptible to TNF-induced cell death