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Genetic research has identified markers associated with varying responses to GLP-1 therapy, including variants in the GLP1R gene, GIPR gene, and metabolic regulators like FTO and MC4R

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Additionally, significant changes were noted in the composition of the bone matrix of these animals, with decreased enzymatic cross-linking of the collagenous matrix, which clearly worsened the compromised biomechanical strength and contributed to the observed bone fragility [44]

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