The effect was due to accelerated angiogenesis but not vasodilation, since there was no significant difference of the blood flow in tails and non-ischemic hind limb between the control and BPC 157 groups
A single exposure (A) causes a small decrease (less than 3% of normal), in both blood and cytosolic GSH and Cys concentrations, with levels returning to above 99% of normal for all species within the simulated time-span
Acknowledgements This work was supported by grants from the Deutsche Forschungsgemeinschaft (SFB 877, Project B2, and Cluster of Excellence Inflammation at Interfaces, EXC306-PMTP1 and EXC306-PWTP2, to D
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