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Haskell, C
On the other hand, Vitamin C is important in limiting free radical damage in the aqueous phase of the cell, and cellular vitamin C status may be linked to the mechanisms involved in quenching cellular reactive oxygen species
B B cha nhiu cht bo khng bo ha n lnh mnh, vitamin E v c bit l glutathione
OS can be triggered by free radicals or other oxidant species produced by either exogenous processes (environmental pollutants, drugs, viruses, bacteria, ionizing radiations, heavy metals, poor diet, and smoking) [3,4,5,6,7,8,9,10,11] or endogenous processes (such as immune cell activation, inflammation, ischemia, infection, cancer, and aging) [12,13,14,15,16,17,18,19]
Garcia-Navarro, A
This means that melanocytes exposed to oxidative stress may undergo necroptosis when the RIP1 pathway is activated [37, 65]
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