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It plays a crucial role in postprandial glucose regulation and satiety signaling that is entirely separate from the incretin system

closer glucose monitoring is prudent

The peptides mechanism of action, however, remains only partially understood, and its receptor system has yet to be definitively identified

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2, in c-Met signaling, overexpression of the c-Met receptor and HGF results in sustained PI3K/Akt/mTOR and MAPK signaling activation through Gab1 and mutation of MET kinase domain, which are associated with therapy resistance [87]

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