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Mechanistically, scientific literature suggests BPC 157 may influence several repair pathways at once, including signaling involved in new blood vessel formation (angiogenesis), nitric oxide-related pathways (important for blood flow and tissue signaling), and growth-factor activity that supports cell migration and remodeling during healing

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Enzymes, pH fluctuations, and the liver's first-pass metabolism can all dramatically reduce the bioavailability of a compound

This leaves a big gap in our knowledge of the dystonic syndromes still to be characterized from an electrophysiologic standpoint