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Mitochondria, which are structures within the cells of the RPE that act as energy powerhouses, are particularly susceptible to ROS-induced damage [17]
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Studies in the mouse model, which closely reproduces the human condition, have shown that hepatotoxicity is initiated by formation of a reactive metabolite N -acetyl- p -benzoquinone imine (NAPQI), which depletes cellular glutathione and forms protein adducts on mitochondrial proteins
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