This releases platelet-derived growth factor (PDGF) , vascular endothelial growth factor (VEGF) , epidermal growth factor (EGF) , and fibroblast growth factor (FGF)
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This observation prompted the authors to consider whether the protective mechanism of A1AT involves not only the direct inhibition of NE activity but also an anti-inflammatory effect that prevents the release of TNF- alpha and subsequent infiltration of inflammatory cells
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