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Although the mechanisms by which HRT promotes neuroprotection are mostly unknown, it has been proposed that signaling pathways mediated by both classical estrogen receptors and GPER-1 are involved in the anti-oxidant and anti-inflammatory effects of estrogens in nervous tissue (Unfer et al., 2015

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13 , e31 (2011)

When collecting highly redundant data at a wavelength close to the copper absorption edge, residual S-atom positions could also be located in log-likelihood-gradient maps and used to improve the phases

The adverse metabolic effects of branched-chain amino acids are mediated by isoleucine and valine

References Billon, C., Sitaula, S., Banerjee, S., Welch, R., Elgendy, B., Hegazy, L.,