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Next, the trans -2 double bond is hydrated stereospecifically by an enoyl-CoA hydratase to form an L3hydroxyacyl-CoA, which is in turn oxidized by a hydroxyacyl-CoA dehydrogenase to yield a 3-ketoacyl-CoA with nicotinamide adenine dinucleotide (NAD) reduced to NADH

Upon reperfusion, the sudden influx of oxygen leads to a burst of ROS production, exacerbating cellular damage

As a consequence of this article, we included in our clinical reports a recommendation to check B12 levels prior to the next injection to clarify status

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Therefore, finding the disturbed metabolic pathways could help us to better understand the pathogenesis of PENs-induced AD