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Pro-inflammatory signaling and Ca 2+ overload can activate kinases that phosphorylate DRP1 and other fission-related proteins, thereby promoting mitochondrial fragmentation, m loss, and increased ROS generation ( The interaction between mitochondria and the cytoskeleton also influences the spatial organization of other key processes in AP, including autophagosome trafficking, lysosomal fusion, and immune cell recruitment to the injured pancreas

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These results are consistent with prior studies showing that statins inhibit selenoprotein synthesis and statin-induced cell death has features of ferroptosis and may be rescued by ferroptosis inhibitors although this is universally consistent for all statins [11, 4547]