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We observed only the full length SnCE1 being lysine acetylated whereas the autoproteolytically processed SnCE1 is not acetylated, which supports the conclusion that SnCE1 deacetylation is a prerequisite for proteolytic processing of SnCE1 (Fig

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The whole group of are formed by adding GSH to LTA 4 , which gives rise to LTC 4 and, when the GSH is split, to LTD 4 , LTE 4 and LTF 4

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Hypoxia increases mitochondrial ROS formation and leads to enhanced infiltration of activated inflammatory cells (71)

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