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An OSCC subgroup termed Epi_2 over-expresses the cuproptosis repressor CDKN2A and displays heightened glycolysis, reinforced glutathione metabolism, and activated hypoxia signaling, which collectively enable the tumor to evade cuproptosis

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Multiple myeloma cells frequently overproduce immunoglobulins subunits and have become the target of the first clinical UPR-targeted therapy to impair ERAD with the proteasome inhibitor, bortezomib (Bross et al

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