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Propionyl-L-carnitine (200 mg/kg, p.o., drinking water) increases NO synthesis by enhancing eNOS expression in spontaneously hypertensive rats [8]

Aside from this possible therapeutic effect of riboflavin, these findings could also introduce the riboflavin-dependent mechanism by which cells could protect themselves from oxidative damage in normal conditions ( Reference Christensen 60 ) and provide further support for the antioxidant nature of riboflavin

GST M2-2 is a prostaglandin E synthase in the brain cortex [8] and rat GST A1-1 and GST A3-3 catalyze the reduction of PGH 2 to PGF 2