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In experimental colitis models, ERS inhibitors such as sodium phenylbutyrate can significantly improve intestinal inflammation, indicating the important role of the ER stress PERK axis in IBD (101)

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We remark that one would not expect the contributions of the two metabolites to GSH synthesis to be strictly proportional to their inputs since they are used in other reactions and the system is highly non-linear

The underlying driver is chronic inflammationspecifically, dysregulated immune signaling involving TNF-, interleukins (IL-17, IL-23), and NF-B pathways