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Secretion of defensins can be constitutive or induced by prokaryotic antigens through a Toll-like-receptor mediated process [133]
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The compound's development represents years of medicinal chemistry optimization, resulting in a 10-fold improvement in potency over parent compounds while maintaining excellent safety margins in preclinical studies
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The proposed mechanism involves maintaining synaptic vesicle integrity, competing for receptor sites, and potentially substituting for depleted neurotransmitters
BPC-157 supports tissue repair and gut integrity
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