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In rheumatoid arthritis, the mTOR pathway inhibits synovial fibroblast ferroptosis by increasing GPX4 expression [70]

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49 , who reported that conversion of the N-terminal histidine (His) of Ex-4 to phenylalanine (Phe) resulted in reduced GLP-1R-mediated internalization and prolonged signal response

Several studies have reported that seizures can activate the hif-1a/HO-1 pathway, leading to abnormal iron metabolism and increased Fe 2+ accumulation in hippocampal neurons