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Abstract Background: Primary Sjogrens syndrome (pSS) is a prototypical systemic autoimmune disease characterised by lymphocyte infiltration and immune-complex deposition in multiple organs

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a G protein-coupled receptor) agonist exendin-4 suppressed -aminobutyric acid receptor (GABAR)-mediated currents through GLP-1Rs in isolated rat retinal ganglion cells (GCs)