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Glutathione functions as the primary intracellular reducing agent, maintained predominantly in its reduced (GSH) form by glutathione reductase using NADPH as a cofactor

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Phase 2 and mechanistic studies report reduced hunger perception, delayed gastric emptying, and effects on postprandial glucoregulatory hormones consistent with amylin signalling, though exact glucagon findings should be attributed to the original trial reports [2]