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The priming signal involves the upregulation of NLRP3 expression and the synthesis of proinflammatory cytokines such as TNF- and IL-1, typically mediated by nuclear factor kappa B activation in response to inflammatory stimuli

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Mitochondrial ion accumulation also contributes to the impact of antioxidant therapies [78, 79] that reduce the release/impact of mitochondrial ROS after TBI