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The experimental trail included a 26-week treatment period, a dose-escalation period of up to 6 weeks, and a 6-week follow-up period after the end of treatment

P = 0.63)

Reduced concentrations of N-acetyl aspartate and total creatine in the basal ganglia are markers of neuronal loss and impaired energy metabolism, which correlate with cognitive dysfunction and motor deficits (Nordahl et al., 2005)

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Newer studies cite even more benefits of these medications, reducing obesity-related disorders like sleep apnea and other metabolic dysfunctions