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Atrial AMP-activated protein kinase is critical for prevention of dysregulation of electrical excitability and atrial fibrillation
A mild increase in nutrient signaling to mTORC1 in mice leads to parenchymal damage, myeloid inflammation and shortened lifespan
The same result may be achieved by redistribution of charged residues to align along one side of the C-terminal alpha helix, with predominantly hydrophobic amino acids on the opposite side ( The juvenile pig was chosen as a large animal model because the pig has a small intestine closer in physiology to humans than any other common animal models
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