-GT is located at luminal endothelial cell membranes of these organs with its active site on the extracellular surface of the membrane rightly positioned for the cleavage of GSH from the blood circulation (Fig
120 MlleM.EschenkoO.GaisS.SaraS
50 Local SDF-1 release increased the recruitment of systemically infused bone marrow-derived MSCs by 700% in a mouse model of myocardial infarction (MI), 51 suggesting that cartilage repair scaffolds releasing SDF-1 may perform synergistically with MSCs from marrow recruitment or intra-articular injections
In macrophages, increased HO-1 expression shows the ability to convert mobilized M1 macrophages into alternately activated M2 macrophages with anti-inflammatory effects, which is essential for the wound healing phase [127]
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