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The effect of thyrotoxicosis on adrenocortical reserve

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MTHFR promotes the synthesis of folate into methylenetetrahydrofolate, which participates in the methionine cycle and gene methylation process [1, 15, 16]

Furthermore, SCFAs activate GPR43, leading to blocking IB phosphorylation and degradation, which inhibits NF-B activity and reduces inflammatory gene expression ( Figure 3 ) [88]