A novel beta-catenin-binding protein inhibits beta-catenin-dependent Tcf activation and axis formation
When bound to copper, the GHK peptide becomes highly active in cellular communication pathways involved in regeneration and structural maintenance
Isayenkov SV, Maathuis FJ
[DOI] [PubMed] [Google Scholar] 41.Verma S., Singh A., Mishra A
Beyond classical antioxidant signaling, GSH interfaces with additional pathways, including Hippo-Yes1 associated transcriptional regulator (YAP1) signaling to regulate GPX4 and SLC7A11 expression, 159,160 mechanistic target of rapamycin kinase (mTOR) signaling to influence GPX4 protein synthesis through cysteine metabolism, 161 and NF-B signaling to restrain inflammation-associated ROS production 162
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