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50,51 In a recent study, MSCs enhanced autophagy and exerted a neuroprotective effect by modulating amyloid- clearance in an AD mouse model

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In reaction to AD, astrocytes experience a shift in profile, one that is reactive and pro-inflammatory while losing homeostatic roles thus disrupting BBB integrity, ion and neurotransmitter buffering, and energy metabolism (Liddelow et al., 2017

Microglia-specific knock-out of NF-B/IKK2 increases the accumulation of misfolded -synuclein through the Inhibition of p62/sequestosome-1-dependent autophagy in the rotenone model of parkinsons disease

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