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Although deletion of CARS2 in mice is embryonic lethal, mice with a heterozygous knockout of CARS2 showed approximately 50% lower levels of supersulfides compared with wild-type mice, indicating a predominant role of mitochondrial CARS2 in supersulfide production (15) ( Figure 2 )

Loss of native -synuclein multimerization by strategically mutating its amphipathic helix causes abnormal vesicle interactions in neuronal cells

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