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R.NonakaT.HasegawaM.et al

The human tripeptide GHK and tissue remodelling

It was identified during pharmacokinetic investigations of MT-2 as the primary active metabolite formed following in-vivo hydrolysis of the MT-2 acetyl cap, resulting in a free N-terminal amine in place of the N-acetyl group

In vitro, synovial fibroblasts exposed to GSH reduced IL-6 secretion, suggesting a retreat of synovial inflammation [6]

Trk receptor activation has variable downstream specificity, and significant cross-talk is observed among the sites of action in these three pathways