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This indicates that survival is critically dependent upon GSH, and therefore is not being sustained by an alternative Trx-disulfide reductase, such as by repurposing of either the mitochondrial TrxR2 or the testis-specific Tgr (TrxR3) [48, 109]
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Nonspherocytic hemolytic anemia (NSHA) NSHA is a member of a group of inherited disorders in which mutations or deficiencies in specific enzymes or proteins involved in red blood cell metabolism disrupt normal cell function and lead to hemolysis
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