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Genetic polymorphisms in GSTs, particularly the GSTM1 and GSTT1 null genotypes, significantly influence SFNs metabolic fate and clinical efficacy

Similarly, the knockdown (KD) of the mitophagy gene in HeLa cells resulted in decreased mitochondrial membrane potential and substantially reduced GSH content [17]

Glutathione is usually present as reduced form (GSH), but GSH is converted into its oxidized form (GSSG) by stimulation such as oxidative stress

[4] One common method is the DTNB-glutathione reductase recycling assay

No biotechnology or genetically modified organisms are involved

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