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Caption: Plasma GHK-Cu concentrations decline by more than 60 percent between ages 20 and 60, a trajectory that tracks closely with the loss of regenerative capacity
Tendinopathy causes a decrease in collagen in the injured tendon, but it actually causes an increase in proteoglycans and GAGs, such as chondroitin sulfate and hyaluronan.[7,8,13,18,40] Gluscosamine sulfate and chondroitin sulfate might help build proteoglycans, but since the levels of GAGs and proteoglycans are already abnormally high in tendinopathy, you wouldn't expect oral supplementation of glucosamine and chondroitin to help
Short-term folic acid supplementation induces variable and paradoxical changes in plasma homocyst(e)ine concentrations
Many autoimmune conditions have gut involvement, whether through intestinal permeability (leaky gut), microbiome disruption, or direct intestinal immune activation
What makes the story even more complicated is that GH has concentration-dependent effects on tissues, as there are indications that one needs less GH to stimulate gluconeogenesis and glycolysis versus IGF-1 generation [34,38]
