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Figure 3 Hyperhomocysteinemia-associated hypertension is gaining increasing attention in clinical diagnosis and treatment, as it is also a risk factor for atherosclerotic cardiovascular disease (36)

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The TXN system suppresses ferroptosis through multiple mechanisms: by supplying reducing equivalents to PRDXs, facilitating cystine-to-cysteine reduction to support GSH synthesis, and acting as an independent ferroptosis suppressor (Fig