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For instance, two siblings both harboring the same mitochondrial mutation causing Lebers hereditary optic neuropathy had markedly different clinical courses: the brother exhibited typical progressive optic neuropathy whereas the sister suffered from a relapsingremitting MS-like illness (Joshi and Kermode, 2019)

Oxeiptosis driven by AIFM1 does not necessitate the migration of AIFM1 from the mitochondria to the nucleus, a distinctive feature that sets oxeiptosis apart from parthanatos [74]

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