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In neuronal cell cultures, GHK-Cu demonstrates remarkable neuroprotective effects, reducing oxidative damage by 55% and enhancing cell survival under stress conditions by 40% compared to control administrations

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The role of IGF-II should be viewed in the light of Efstradiadis series of knock-out experiments in mice (21) where the Igf2 ko mice had the same degree of fetal growth retardation as the igf1 ko which demonstrates that IGF-II is a paracrine/autocrine and not an endocrine hormone