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10.1093/jexbot/52.354.145 119 ScandaliosJ

Here we reviewed only evidence for mitochondrial defects in schizophrenia

Further, glucose hypermetabolism was identified by PET scans with 18F-FDG in the basal ganglia, and this correlated with several forms of encephalitis and is often coupled to hypometabolism in other brain regions (Rey et al., 2012

Protection of the BBB, the inhibition of the elements of the brain microvascular damage such as impairment of tight junctions, increased vesicular transcytosis, production of various cytokines, chemokines and reactive oxygen species offers novel therapeutic targets [11]

ButlerCLHickeyMJJiangNZhengYGjertsonDZhangQet alDiscovery of non-HLA antibodies associated with cardiac allograft rejection and development and validation of a non-HLA antigen multiplex panel: from bench to bedside

Understanding the mechanisms underlying the vulnerability of SNpc dopaminergic neurons in PD remains critical for developing new therapeutic approaches to halt or slow the progression of this debilitating disease